Author

K. S. Lang

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Organism:bacteria (other)· 1 Assay:bulk RNA-seq· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

A. Bazarna· 1 shared A. Günther· 1 shared A. Paschen· 1 shared A. W. Chan· 1 shared A. Westendorf· 1 shared B. Jeon· 1 shared B. Kleuser· 1 shared B. Scheffler· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperCancer Biology

A. Bazarna

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Development of fluoroquinolone resistance through antibiotic tolerance in Campylobacter jejuni

M. Park, J. Kim, J. Feinstein, K. S. Lang, S. Ryu, B. Jeon

This study shows that antibiotic tolerance in Campylobacter jejuni induced by ciprofloxacin and tetracycline leads to rapid development of fluoroquinolone resistance.

bioRxiv · May 21, 2022Microbiology

The acid ceramidase/ceramide axis controls parasitemia in Plasmodium yoelii-infected mice by regulating erythropoiesis

A. Günther, M. Hose, F. Schumacher, B. Kleuser, K. Matuschewski, K. S. Lang, E. Gulbins, J. Buer, A. Westendorf, W. Hansen

This study shows that acid ceramidase deficiency reduces parasitemia in Plasmodium yoelii-infected mice by impairing erythropoiesis and T cell responses.

bioRxiv · Mar 8, 2022Microbiology

Progranulin promotes immune evasion of pancreatic adenocarcinoma through regulation of MHCI expression

P. F. Cheung, J. Yang, K. Krengel, K. Althoff, C. Yip, E. H. Siu, L. W. Ng, K. S. Lang, L. Cham, D. R. Engel, C. Soun, I. Cima, B. Scheffler, J. K. Striefler, M. Sinn, , J. T. Siveke

This study shows that tumor-derived progranulin (PGRN) promotes immune evasion in pancreatic ductal adenocarcinoma by reducing MHC class I expression, and blocking PGRN enhances immune response and slows tumor progression.

bioRxiv · Feb 24, 2021 · PreprintCancer Biology