Author

Kangping Du

University of Virginia

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:database / resource· 1

Frequent Co-Authors

Aaron Nakasone· 1 shared Basel Maher Al-BarghouthiiD· 1 shared Bhavya SenwariD· 1 shared Charles R. FarberiD· 1 shared Cheryl L. Ackert‐BicknelliD· 1 shared David L. BrautiganiD· 1 shared Elise Feng‐i MorganiD· 1 shared Gina M. CalabreseiD· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperGenomics

Aaron Nakasone

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2 papers

Recent·Most cited

Break-induced replication drives large-scale genomic amplifications in cancer cells

Mouadh Benamar, Rebeka Eki, Kangping Du, Tarek Abbas

This study shows that DNA double-strand breaks induce large-scale genomic amplifications in cancer cells through a break-induced replication mechanism.

bioRxiv · Aug 27, 2024 · PreprintCancer Biology

Transcriptome-wide Association Study and eQTL colocalization identify potentially causal genes responsible for bone mineral density GWAS associations

Basel Maher Al-Barghouthi, Will T. Rosenow, Kangping Du, Jinho Heo, Robert D. Maynard, Larry D. Mesner, Gina M. Calabrese, Aaron Nakasone, Bhavya Senwar, Louis C. Gerstenfeld, James Mitchell Larner, Virginia L. Ferguson, Cheryl L. Ackert‐Bicknell, Elise Feng‐i Morgan, David L. Brautigan, Charles R. Farber

The paper identifies 512 potentially causal genes for bone mineral density using TWAS and eQTL colocalization, with PPP6R3 validated in mice.

eLife · Nov 17, 2022Peer-reviewedGenomics