Author

Keimpe Weirda

VIB-KU Leuven Center for Brain & Disease Research

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Neuroscience· 1

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

Ben VermaerckeiD· 1 shared Leïla BoubakariD· 1 shared Martyna DitkowskaiD· 1 shared Paula RodrígueziD· 1 shared Pierre VanderhaegheniD· 1 shared Ryohei IwataiD· 1 shared Vincent BoniniD· 1 shared

Activity by Year

2023-2023 · peak 1/yr

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7 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Ben VermaerckeORCID

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1 paper

Recent·Most cited

SYNGAP1 deficiency disrupts neoteny in human cortical neurons in vivo.

Ben Vermaercke, Ryohei Iwata, Keimpe Weirda, Leïla Boubakar, Paula Rodríguez, Martyna Ditkowska, Vincent Bonin, Pierre Vanderhaeghen

This study shows that SYNGAP1 deficiency causes accelerated synaptic development and disrupted neoteny in human cortical neurons transplanted into mice, linking it to ID/ASD.

bioRxiv · Jan 14, 2023 · PreprintNeuroscience