Author

Kelly Cavallo

University of Massachusetts Chan Medical School

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

Samuel M. BehariD· 3 shared Evelyn ChangiD· 2 shared Rocky LaiiD· 2 shared Abiola F. OgunsolaiD· 1 shared Anthony TraniD· 1 shared Gillian BeameriD· 1 shared Tasfia RakibiD· 1 shared Tomoyo Shinkawa· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

8 top collaborators · 19 mutual co-author links

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3 shared papersImmunology

Samuel M. BeharORCID

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3 papers

Recent·Most cited

BCG vaccination elicits protection against Mtb infection mediated by two phases of T cell immunity

Abiola F. Ogunsola, Rocky Lai, Kelly Cavallo, Anthony Tran, Gillian Beamer, Samuel M. Behar

This study shows BCG vaccination protects against Mtb infection in CC042 mice through two T cell-dependent phases: an early innate response and a later effector T cell response.

JCI Insight · Jul 2, 2026Peer-reviewedImmunology

CD226 identifies effector CD8+ T cells during tuberculosis and costimulates recognition of Mycobacterium tuberculosis-infected macrophages

Tomoyo Shinkawa, Evelyn Chang, Tasfia Rakib, Kelly Cavallo, Rocky Lai, Samuel M. Behar

The paper identifies CD226 as a marker distinguishing effector from dysfunctional CD8+ T cells in Mtb infection and shows its role in costimulating recognition of Mtb-infected macrophages.

Cell Reports · Aug 30, 2025Peer-reviewedImmunology

CD4 T cell dysfunction is associated with bacterial recrudescence during chronic tuberculosis.

Evelyn Chang, Kelly Cavallo, Samuel M. Behar

This study investigates CD4 T cell dysfunction in chronic tuberculosis and links it to bacterial recrudescence in mice.

Nature Communications · Mar 17, 2025Peer-reviewedImmunology