Alternative splicing of auxiliary β2-subunits stabilizes Cav2.3 Ca2+ channel activity in continuously active midbrain dopamine neurons
Anita Siller, Nadja T. Hofer, Giulia Tomagra, Nicole Burkert, Simon Heß, Julia Benkert, Aisylu Gaifullina, Desirée Spaich, Johanna Duda, Christina Pötschke, Kristina Vilusic, Eva Maria Fritz, Toni Schneider, Peter Kloppenburg, Birgit Liss, , Jörg Striessnig
The paper shows that alternative splicing of β2-subunits stabilizes Cav2.3 Ca2+ channel activity in midbrain dopamine neurons, contributing to their vulnerability in Parkinson's disease.
eLife · Jul 6, 2022Peer-reviewedNeuroscience