Author

Julia Benkert

Universität Ulm

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

Birgit LissiD· 2 shared Christina Pötschke· 2 shared Johanna Duda· 2 shared Nicole Burkert· 2 shared Aisylu GaifullinaiD· 1 shared Anita SilleriD· 1 shared Desirée Spaich· 1 shared Dominik Wuttke· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersNeuroscience

Birgit LissORCID

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Author profile →

2 papers

Recent·Most cited

Deep learning-based image analysis identifies a DAT-negative subpopulation of dopaminergic neurons in the lateral Substantia nigra.

Nicole Burkert, Shoumik Roy, Max Häusler, Dominik Wuttke, Sonja Müller, Johanna Wiemer, Helene Hollmann, Marvin Oldrati, Jorge Ramírez‐Franco, Julia Benkert, Michael Fauler, Johanna Duda, Jean‐Marc Goaillard, Christina Pötschke, Moritz Münchmeyer, , Birgit Liss

The paper uses deep learning to identify a DAT-negative subpopulation of dopaminergic neurons in the lateral substantia nigra in mouse and human brain sections.

Communications Biology · Nov 10, 2023Peer-reviewedNeuroscience

Alternative splicing of auxiliary β2-subunits stabilizes Cav2.3 Ca2+ channel activity in continuously active midbrain dopamine neurons

Anita Siller, Nadja T. Hofer, Giulia Tomagra, Nicole Burkert, Simon Heß, Julia Benkert, Aisylu Gaifullina, Desirée Spaich, Johanna Duda, Christina Pötschke, Kristina Vilusic, Eva Maria Fritz, Toni Schneider, Peter Kloppenburg, Birgit Liss, , Jörg Striessnig

The paper shows that alternative splicing of β2-subunits stabilizes Cav2.3 Ca2+ channel activity in midbrain dopamine neurons, contributing to their vulnerability in Parkinson's disease.

eLife · Jul 6, 2022Peer-reviewedNeuroscience