Author

L. Poisson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1

Frequent Co-Authors

I. Datta· 2 shared R. Rattan· 2 shared S. Giri· 2 shared A. D. Berezovsky· 1 shared A. deCarvalho· 1 shared A. Munkarah· 1 shared A. Y. Taha· 1 shared C. Mueller· 1 shared

Activity by Year

2023-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersCancer Biology

I. Datta

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Impact of genomic background and developmental state on signaling pathways and response to therapy in Glioblastoma patient-derived cells

A. D. Berezovsky, O. Nuga, I. Datta, K. Bergman, T. Sabedot, K. Gurdziel, S. Irtenkauf, L. Hasselbach, Y. Meng, C. Mueller, E. Petricoin, S. Brown, N. Purandare, S. Aras, T. Mikkelsen, , A. deCarvalho

This study examines how genomic background and developmental state affect signaling pathways and therapy response in GBM patient-derived cells, finding that differentiation increases TMZ resistance and activates DNA-damage interferon responses.

bioRxiv · Mar 14, 2024 · PreprintCancer Biology

Blood-based targeted metabolipidomics reveals altered omega fatty acid-derived lipid mediators in relapsing-remitting multiple sclerosis patients

I. Zahoor, J. Waters, N. Ata, I. Datta, T. Pedersen, M. Cerghet, L. Poisson, S. Markovic-Plese, R. Rattan, A. Y. Taha, J. W. Newman, S. Giri

The paper identifies altered omega fatty acid-derived lipid mediators in RRMS patients using metabolipidomics, suggesting their potential as biomarkers for diagnosis and prognosis.

bioRxiv · Jan 4, 2024 · PreprintBiochemistry

Intermittent Fasting induced ketogenesis inhibits mouse epithelial ovarian tumors by promoting anti-tumor T cell response.

M. P. Udumula, H. Singh, R. Faraz, L. Poisson, N. Tiwari, I. Dimitrova, M. Hijaz, R. Gogoi, M. Swenor, A. Munkarah, S. Giri, R. Rattan

This study shows that intermittent fasting inhibits mouse epithelial ovarian tumors by enhancing anti-tumor T cell responses through metabolic changes and immune modulation.

bioRxiv · Mar 8, 2023 · PreprintCancer Biology