Author

L. Rempel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:flow cytometry· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1

Frequent Co-Authors

F. F. Li· 2 shared F. M. V. Rossi· 2 shared M. Low· 2 shared M. Theret· 2 shared A. Wu· 1 shared C. Eisner· 1 shared C. K. Chang· 1 shared E. Sauge· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersCell Biology

F. F. Li

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2 papers

Recent·Most cited

Mesenchymal Progenitors set the homeostatic inflammatory milieu via the TAK1-NFkB axis.

M. Theret, M. Messing, Z. White, L. W. Henry, L. Rempel, M. Hamer, J. Hashimoto, F. F. Li, J. Brassard, Y. Li, E. Sauge, S. Shin, K. Day, M. Uppal, M. Low, , F. M. V. Rossi

This study shows mesenchymal progenitors regulate the balance between type 1 and type 2 inflammation via the TAK1-NFkB pathway, with implications for inflammatory diseases.

bioRxiv · Dec 11, 2022 · PreprintCell Biology

Targeting fibrosis in the Duchenne Muscular Dystrophy mice model: an uphill battle

M. Theret, M. Low, L. Rempel, F. f. Li, L. W. Tung, O. Contreras, C. K. Chang, A. Wu, H. Soliman, F. M. V. Rossi

The paper screens drugs to block fibrogenic differentiation in Duchenne Muscular Dystrophy mice, but finds no significant improvement in muscle histopathology despite in vitro hits.

bioRxiv · Jan 20, 2021 · PreprintCell Biology