Author

Lalita Ramakrishnan

Primary ORCID profile (0000-0003-0692-5533 · 6 papers)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Lalita RamakrishnaniD6 papers · MRC Laboratory of Molecular Biology

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:imaging· 1 Organism:zebrafish· 1

Frequent Co-Authors

Amit Kumar Verma· 1 shared C.J. Cambier· 1 shared David R. ShermaniD· 1 shared Dinesh Kumar Singhal· 1 shared Harresh Adikesavalu· 1 shared Joseph KeaneiD· 1 shared Kevin B. UrdahliD· 1 shared Kristin N. Adams· 1 shared

Activity by Year

2017-2018 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperMicrobiology

Amit Kumar Verma

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2 papers

Recent·Most cited

MicrobiologybioRxivPreprintAug 3, 2018

Macrophage-induced rifampin tolerance across Mycobacterium tuberculosis lineages is Rv1258c-dependent

Kristin N. Adams, Amit Kumar Verma, Radha Gopalaswamy…·Rafael E. Hernandez

The paper shows that Rv1258c is required for rifampin tolerance in Mtb Lineage 4 strains but not isoniazid tolerance, and that other lineages develop rifampin tolerance except Lineage 2 Beijing strains.

DOI 10.1101/383109

humanexperimental assay

ImmunologybioRxivPreprintJun 7, 2017

Phenolic glycolipid facilitates mycobacterial escape from a microbicidal population of tissue-resident macrophages

C.J. Cambier, Seónadh M. O’Leary, Mary P. O’Sullivan…·Lalita Ramakrishnan

The paper shows that mycobacteria use phenolic glycolipid (PGL) to escape resident macrophages by activating the STING pathway and recruiting monocytes, suggesting PGL-blocking interventions could prevent tuberculosis.

DOI 10.1101/147421