Author

Laszlo F. Locskai

University of Alberta

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 4

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:zebrafish· 4 Research Focus:cell type annotation· 1 Assay:proteomics· 1

Frequent Co-Authors

W. Ted AllisoniD· 4 shared Hadeel AlyenbaawiiD· 3 shared Edward A. BurtoniD· 2 shared Melissa J KinleyiD· 2 shared Samantha A. W. Tan· 2 shared Taylor Gill· 2 shared Alexander H. BurtoniD· 1 shared Holger WilleiD· 1 shared

Activity by Year

2021-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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4 shared papersNeuroscience

W. Ted AllisonORCID

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4 papers

Recent·Most cited

Seizures and tauopathy following neurotrauma are mediated by prion protein and metabotropic glutamate receptor 5

Laszlo F. Locskai, Janelle Nordin, Taylor Gill, Melissa J Kinley, Hadeel Alyenbaawi, W. Ted Allison

This study investigates the role of the mGluR5-PrPC pathway in linking traumatic brain injury to seizures and tauopathy using zebrafish models.

bioRxiv · Sep 27, 2026 · PreprintNeuroscience

JNJ-42153605, a mGluR2 PAM, potentiates Levetiracetam treatments of TBI to mitigate subsequent tau aggregation in a larval zebrafish model

Laszlo F. Locskai, Shayleen Ghassemi, Samantha A. W. Tan, Melissa J Kinley, W. Ted Allison

The paper tests whether anti-epileptic drugs and mGluR2 PAMs applied after TBI can prevent tau aggregation and neurodegeneration in a zebrafish model.

Experimental Neurology · Sep 9, 2026Peer-reviewedNeuroscience

A larval zebrafish model of TBI: Optimizing the dose of neurotrauma for discovery of treatments and aetiology

Laszlo F. Locskai, Taylor Gill, Samantha A. W. Tan, Alexander H. Burton, Hadeel Alyenbaawi, Edward A. Burton, W. Ted Allison

This paper develops a larval zebrafish model of TBI to optimize injury severity for studying treatment and aetiology, observing correlations between injury levels and clinical phenotypes like seizures and tau aggregation.

bioRxiv · Jun 14, 2024 · PreprintNeuroscience

Seizures are a druggable mechanistic link between TBI and subsequent tauopathy

Hadeel Alyenbaawi, Richard Kanyo, Laszlo F. Locskai, Razieh Kamali-Jamil, Michèle G. DuVal, Qing Bai, Holger Wille, Edward A. Burton, W. Ted Allison

This study shows that post-traumatic seizures and dynamin-dependent endocytosis promote tauopathy spread after TBI using a zebrafish model.

eLife · Feb 1, 2021Peer-reviewedNeuroscience