Author

Lianteng Zhi

Thomas Jefferson University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:spatial transcriptomics· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:trajectory inference· 1

Frequent Co-Authors

Yuanxin CheniD· 2 shared Chenbo ZengiD· 1 shared Chenjian LiiD· 1 shared Huaixin Wang· 1 shared Hui ZhangiD· 1 shared Hui ZhangiD· 1 shared Jingyu ZhaoiD· 1 shared Qing Xu· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 20 mutual co-author links

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2 shared papersNeuroscience

Yuanxin ChenORCID

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2 papers

Recent·Most cited

The Human LRRK2-R1441G Mutation Drives Age-Dependent Oxidative Stress and Mitochondrial Dysfunction in Dopaminergic Neurons

Yuanxin Chen, Lianteng Zhi, Shiquan Cui, Huaixin Wang, Chenbo Zeng, Hui Zhang

This study shows that the LRRK2-R1441G mutation causes age-related mitochondrial dysfunction and oxidative stress in dopamine neurons, suggesting calcium channels and uncoupling proteins as therapeutic targets.

Molecular Neurodegeneration · Jul 7, 2026Peer-reviewedNeuroscience

Loss of PINK1 causes age-dependent mitochondrial trafficking deficits in nigrostriatal dopaminergic neurons through aberrant activation of p38 MAPK

Jingyu Zhao, Yuanxin Chen, Lianteng Zhi, Qing Xu, Hui Zhang, Chenjian Li

This study shows that PINK1 loss causes age-dependent mitochondrial trafficking deficits in dopaminergic neurons via p38 MAPK hyperactivation, leading to impaired mitochondrial motility and increased oxidative stress.

bioRxiv · Jul 17, 2024 · PreprintNeuroscience