Author

Louison Pineau

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Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Assay:electronic health records· 2 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Organism:mouse· 1 Organism:rat· 1

Frequent Co-Authors

Alfonso RepresaiD· 1 shared Aurélie Montheil· 1 shared Carlos CardosoiD· 1 shared Emilie Pallesi‐PocachardiD· 1 shared Emmanuelle Buhler· 1 shared Françoise Watrin AiD· 1 shared Hélène Becq· 1 shared Laurent AniksztejniD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Alfonso RepresaORCID

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2 papers

Recent·Most cited

Early and transient increase in cortical pyramidal cell excitability and delayed alteration of evoked synaptic transmission and t-SNARE proteins content in the hippocampus and neocortex of neonatal and juvenile STXBP1 heterozygous mice

Louison Pineau, Mélanie Brosset-Heckel, Hélène Becq, Emilie Pallesi‐Pocachard, Aurélie Montheil, Najoua Biba, Mathieu Milh, Pierre‐Pascal Lenck‐Santini, Laurent Aniksztejn

This study examines the developmental stage-dependent effects of Munc18.1 deficiency on neuronal excitability and synaptic transmission in STXBP1 heterozygous mice.

Frontiers in Cellular Neuroscience · May 18, 2026Peer-reviewedNeuroscience

Pathogenic MTOR somatic variant causing focal cortical dysplasia drives hyperexcitability via overactivation of neuronal GluN2C NMDA receptors

Louison Pineau, Emmanuelle Buhler, Sarah Tarhini, Š. Bauer, Valérie Crépel, Françoise Watrin A, Carlos Cardoso, Alfonso Represa, Pierre Szepetowski, Nail Burnashev

This study shows that a somatic MTOR variant in FCD type II causes hyperexcitability through overactivation of GluN2C NMDA receptors in rat neurons.

bioRxiv · Dec 1, 2023 · PreprintNeuroscience