Author

M. D. Paraskevopoulou

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Bioengineering· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1

Frequent Co-Authors

E. Chavdoula· 2 shared P. N. Tsichlis· 2 shared S. Singh· 2 shared V. Anastas· 2 shared A. A. Kalinin· 1 shared A. Apostolou· 1 shared A. Banerjee· 1 shared A. D. Kaba· 1 shared

Activity by Year

2020-2022 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersCancer Biology

E. Chavdoula

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Pharmacology & ToxicologybioRxivJun 8, 2022

Integrating inflammatory biomarker analysis and artificial intelligence-enabled image-based profiling to identify drug targets for intestinal fibrosis

S. Yu, A. A. Kalinin, M. D. Paraskevopoulou…·S. R. Horman

The paper identifies potential drug targets for intestinal fibrosis by combining image-based morphology analysis with AI and inflammatory biomarker measurements.

DOI 10.1016/j.chembiol.2023.06.014

humanproteomics

𝕏 PostBluesky

BioengineeringbioRxivPreprintAug 28, 2020

A Micro-engineered Human Colon Intestine-Chip Platform to Study Leaky Barrier

A. Apostolou, R. Panchakshari, A. Banerjee…·K. Karalis

This paper introduces a Colon Intestine-Chip platform to study leaky gut by combining organoids and Organ-on-Chip technologies, showing its response to inflammatory cytokines and IL-22.

DOI 10.1101/2020.08.28.271759

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivJul 14, 2020

IWS1 phosphorylation promotes cell proliferation and predicts poor prognosis in EGFR mutant lung adenocarcinoma patients, through the cell cycle-regulated U2AF2 RNA splicing.

G. I. Laliotis, E. Chavdoula, M. D. Paraskevopoulou…·P. N. Tsichlis

The paper was withdrawn due to concerns about the validity of the reported splicing mechanism and data integrity.

DOI 10.1038/s41467-021-24795-1

bulk RNA-seqexperimental assay

𝕏 PostBluesky

Cancer BiologybioRxivPreprintMay 21, 2020

KDM2B Silencing Elicits a Paracrine Mechanism Which Destabilizes SLUG, Promoting Differentiation of Basal-Like Breast Cancer Cells.

E.-A. Fraile, B. Soysal, V. Anastas…·P. N. Tsichlis

This paper shows that silencing KDM2B triggers a paracrine mechanism that inactivates GSK3, leading to SLUG degradation and differentiation of basal-like breast cancer cells.

DOI 10.1101/2020.05.21.109819

humanproteomics

𝕏 PostBluesky