Author

M. Hershfinkel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1 Assay:imaging· 1 Research Focus:metabolism & bioenergetics· 1 Approach:microscopy & imaging analysis· 1

Frequent Co-Authors

I. Sekler· 2 shared A. Stavsky· 1 shared E. A. Assali· 1 shared G. E. Stutzmann· 1 shared I. Savic Azoulay· 1 shared M. Rozenfeld· 1 shared M. Taha· 1 shared O. Kofman· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersPhysiology

I. Sekler

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2 papers

Recent·Most cited

NCLX controls hepatic mitochondrial Ca2+ extrusion and couples hormone-mediated mitochondrial Ca2+ oscillations with gluconeogenesis

M. Taha, E. A. Assali, G. E. Stutzmann, O. S. Shirihai, M. Hershfinkel, I. Sekler

This study identifies NCLX as the primary mitochondrial Ca2+ extruder in hepatocytes, linking hormone-mediated mitochondrial Ca2+ oscillations to gluconeogenesis.

bioRxiv · Feb 9, 2024Physiology

The mitochondrial Na+/Ca2+ exchanger, NCLX, mediates PDE2 dependent neuronal survival and learning

M. Rozenfeld, I. Savic Azoulay, T. Ben Kasus Nissim, A. Stavsky, M. Hershfinkel, O. Kofman, I. Sekler

This study links PDE2 inhibition to mitochondrial Ca2+ efflux via NCLX, enhancing neuronal survival and learning.

bioRxiv · Jul 8, 2021 · PreprintPhysiology