Author

M. J. Harriff

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 2 Assay:bulk RNA-seq· 1 Assay:proteomics· 1

Frequent Co-Authors

C. M. Heisler· 2 shared D. M. Lewinsohn· 2 shared M. E. Huber· 2 shared T. N. Lust· 2 shared A. H. Worley· 1 shared A. Nellore· 1 shared A. Olive· 1 shared A. Worley· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersImmunology

C. M. Heisler

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Author profile →

3 papers

Recent·Most cited

Delivery of loaded MR1 Monomer Results in Efficient Ligand Exchange to Host MR1 and Subsequent MR1T cell activation

C. A. Kulicke, G. M. Swarbrick, N. A. Ladd, M. Cansler, M. Null, A. Worley, C. Lemon, T. Ahmed, J. Bennett, T. N. Lust, C. M. Heisler, M. E. Huber, J. R. Krawic, L. M. Ankley, S. K. McBride, , M. J. Harriff

The paper demonstrates that delivering MR1 monomers loaded with 5-OP-RU enables ligand exchange to host MR1, leading to MR1T cell activation.

bioRxiv · Jul 11, 2022Immunology

Chronic obstructive pulmonary disease and cigarette smoke exposure lead to dysregulated MAIT cell activation by bronchial epithelial cells

M. E. Huber, E. Larson, T. N. Lust, C. M. Heisler, M. J. Harriff

This study shows that chronic obstructive pulmonary disease and cigarette smoke exposure alter MAIT cell activation by bronchial epithelial cells, leading to dysregulated immune responses.

bioRxiv · Feb 28, 2022 · PreprintImmunology

Alternative Splicing of MR1 regulates antigen presentation to MAIT cells

G. A. Narayanan, A. Nellore, J. Tran, A. H. Worley, E. W. Meermeier, E. Karamooz, M. Huber, R. Kurapova, F. Tafesse, M. J. Harriff, D. M. Lewinsohn

This study investigates how alternative splicing of MR1 affects antigen presentation to MAIT cells, finding that only MR1A can activate MAIT cells while MR1B inhibits their activation.

bioRxiv · Jul 11, 2019Immunology