Author

M. J. Wolf

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Physiology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 4 Approach:experimental assay· 4 Research Focus:cell signaling & trafficking· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:clustering· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Young· 2 shared A. Borchardt· 1 shared A. G. Wolpe· 1 shared A. Hahn· 1 shared A. Reichert· 1 shared A. Schaenzer· 1 shared A. Szagdaj· 1 shared A. V. Busley· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersPhysiology

A. Young

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Pannexin 1 Channels Control Cardiomyocyte Metabolism and Neutrophil Recruitment During Non-Ischemic Heart Failure

C. M. Pavelec, A. Young, H. L. Luviano, E. E. Orrell, A. Szagdaj, N. Poudel, A. G. Wolpe, S. H. Thomas, S. Yeudall, C. Upchurch, M. D. Okusa, B. E. Isakson, M. J. Wolf, N. Leitinger

This study shows that PANX1 deficiency in cardiomyocytes reduces glycolytic metabolism and protects against cardiac hypertrophy in non-ischemic heart failure by decreasing neutrophil recruitment.

bioRxiv · Dec 29, 2023 · PreprintPhysiology

Alteration of myocardial structure and function in RAF1associated Noonan syndrome: Insights from cardiac disease modeling based on patient-derived iPSCs

S. Nakhaei-Rad, F. Bazgir, J. Dahlmann, A. V. Busley, M. Buchholzer, F. Haghighi, A. Schaenzer, A. Hahn, S. Koetter, D. Schanze, R. Anand, F. Funk, A. Borchardt, A. V. Kronenbitter, J. Scheller, , M. R. Ahmadian

This study uses patient-derived iPSCs to model RAF1-associated Noonan syndrome and identifies sarcomere structural abnormalities linked to cardiac dysfunction.

bioRxiv · Jan 22, 2022 · PreprintCell Biology

Ablation of endogenously cycling adult cardiomyocytes worsens myocardial function after injury

L. A. Bradley, A. Young, H. O. Billcheck, M. J. Wolf

bioRxiv · Sep 21, 2020Cell Biology

Suppression of store-operated calcium entry causes dilated cardiomyopathy of the Drosophila heart

J. T. Smyth, M. J. Wolf, C. E. Petersen

This study shows that suppressing store-operated calcium entry in Drosophila hearts leads to dilated cardiomyopathy, reduced contractility, and decreased animal viability.

bioRxiv · Oct 25, 2019Physiology