Author

M. Luetge

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Organism:mouse· 2 Assay:scRNA-seq· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

B. Ludewig· 3 shared H.-W. Cheng· 3 shared A. E. Denton· 1 shared A. Geng· 1 shared A. L. Fletcher· 1 shared A. Waltenspuehl· 1 shared C. Bernsmeier· 1 shared C. J. Weston· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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3 shared papersImmunology

B. Ludewig

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Author profile →

3 papers

Recent·Most cited

Distinct circulating monocytes up-regulate CD52 and sustain innate immune function in patients with cirrhosis unless acute decompensation emerges

A. Geng, R. G. Brenig, J. Roux, M. Luetge, H.-W. Cheng, E. E. Flint, M.-A. Meier, O. T. Pop, P. Kuenzler-Heule, M. McPhail, S. Soysal, D. Semela, M. Heim, C. J. Weston, B. Ludewig, C. Bernsmeier

The paper identifies CD52-expressing monocytes in cirrhosis that enhance innate immunity, but their loss during acute decompensation correlates with infection and poor outcomes.

bioRxiv · Apr 3, 2024 · PreprintImmunology

TYPE I INTERFERON-DEPENDENT CELLULAR REMODELING DURING ACUTE PULMONARY MHV INFECTION

S. Grabherr, A. Waltenspuehl, M. Luetge, L. Buechler, H.-W. Cheng, K. Zielinska, S. Caviezel-Firner, P. Krebs, B. Ludewig, N. B. Pikor

This study investigates the role of type I interferon in limiting viral spread and inflammation during pulmonary MHV infection, showing its necessity for immune cell maturation and viral containment.

bioRxiv · Feb 1, 2022 · PreprintImmunology

Fibroblastic reticular cells provide a supportive niche for lymph node-resident macrophages

J. D'Rozario, K. Knoblich, M. Luetge, C. Perez Shibayama, H.-W. Cheng, Y. O. Alexandre, D. Roberts, J. Campos, J. Astarita, E. Dutton, M. Suliman, A. E. Denton, S. J. Turley, R. L. Boyd, S. Mueller, , A. L. Fletcher

This study identifies fibroblastic reticular cells (FRCs) as essential for supporting lymph node-resident macrophages through CSF1 and CSF1R signaling.

bioRxiv · Nov 24, 2021Immunology