Author

M. Nasu

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:trajectory inference· 1

Frequent Co-Authors

A. Kwag· 1 shared A. UMEZAWA· 1 shared C. Williams· 1 shared F. Revetta· 1 shared G. Hiremath· 1 shared H. Hatsuse· 1 shared H. Orita· 1 shared J. Chaparro· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCancer Biology

A. Kwag

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Eosinophils exert direct and indirect anti-tumorigenic effects in the development of esophageal squamous cell carcinoma

J. Jacobse, Z. Aziz, L. Sun, J. Chaparro, J. M. Pilat, A. Kwag, M. Buendia, M. Wimbiscus, M. Nasu, T. Saito, S. Mine, H. Orita, F. Revetta, S. Short, M. K. Washington, , Y. A. Choksi

This study shows eosinophils reduce esophageal squamous cell carcinoma by releasing reactive oxygen species and inhibiting IL-17 pathways.

bioRxiv · Jun 1, 2023 · PreprintCancer Biology

Gorlin syndrome-induced pluripotent stem cells form medulloblastoma with loss of heterozygosity in PTCH1

Y. Ikemoto, T. Miyashita, M. Nasu, H. Hatsuse, K. Kajiwara, K. Fujii, T. Motojima, M. Toyoda, A. UMEZAWA

This study shows that Gorlin syndrome patient-derived iPSCs form medulloblastoma in mice, with one tumor showing PTCH1 loss of heterozygosity.

Aging · May 21, 2020Peer-reviewedCell Biology