Author

M. R. Scribner

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Microbiology· 4

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 4 Assay:whole-genome sequencing· 3 Organism:bacteria (other)· 2 Research Focus:drug response / drug discovery· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

V. S. Cooper· 4 shared A. Santos-Lopez· 2 shared C. W. Marshall· 2 shared A. C. Stephens· 1 shared A. R. Richardson· 1 shared C. Deitrick· 1 shared C. J. McManus· 1 shared D. Snyder· 1 shared

Activity by Year

2019-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

4 shared papersMicrobiology

V. S. Cooper

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

The nutritional environment is sufficient to select coexisting biofilm and quorum-sensing mutants of Pseudomonas aeruginosa

M. R. Scribner, A. C. Stephens, J. L. Huong, A. R. Richardson, V. S. Cooper

This study shows that nutrient conditions and biofilm selection alone can drive the evolution of Pseudomonas aeruginosa mutants with clinical phenotypes like increased biofilm and altered quorum sensing.

bioRxiv · Sep 1, 2021Microbiology

Quantitative mapping of mRNA 3' ends in Pseudomonas aeruginosa reveals a pervasive role for premature 3' end formation in response to azithromycin

S. Konikkat, M. R. Scribner, R. Eutsey, N. L. Hiller, V. S. Cooper, C. J. McManus

This study uses 3pMap to analyze mRNA 3' ends in Pseudomonas aeruginosa under azithromycin treatment, revealing premature 3' end formation as a key response mechanism.

bioRxiv · Jun 16, 2020 · PreprintMicrobiology

Parallel evolution of tobramycin resistance across species and environments

M. R. Scribner, A. Santos-Lopez, C. W. Marshall, C. Deitrick, V. S. Cooper

This study identifies parallel mutations in fusA1 and ptsP across two bacterial species under tobramycin selection, showing predictable resistance mechanisms despite environmental and genetic differences.

bioRxiv · Sep 6, 2019Microbiology

Evolutionary pathways to antibiotic resistance are dependent upon environmental structure and bacterial lifestyle

A. Santos-Lopez, C. W. Marshall, M. R. Scribner, D. Snyder, V. S. Cooper

This study shows that biofilm lifestyle in Acinetobacter baumannii leads to different genetic pathways and fitness trade-offs in antibiotic resistance evolution compared to planktonic growth.

bioRxiv · Apr 12, 2019Microbiology