Author

M. Ralle

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Pathology· 1 Plant Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1 Organism:plant (other)· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Freywald· 1 shared A. Ribeiro· 1 shared C. Hui· 1 shared D. Bodine· 1 shared D. Strenkert· 1 shared E. Nishibori· 1 shared E.-M. E. Uhlemann· 1 shared F. S. Vizeacoumar· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperBiochemistry

A. Freywald

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

MEMO1 is a Metal Containing Regulator of Iron Homeostasis in Cancer Cells

N. Dolgova, E.-M. E. Uhlemann, M. T. Boniecki, F. S. Vizeacoumar, M. Ralle, M. Tonelli, S. A. Abbas, J. Patry, H. Elhasasna, A. Freywald, F. Vizeacoumar, O. Y. Dmitriev

This study identifies MEMO1 as an iron-binding protein that regulates iron homeostasis in cancer cells through interactions with iron-related proteins and its role in ferroptosis.

bioRxiv · Feb 28, 2023Biochemistry

Single-cell Visualization and Quantification of Trace Metals in Chlamydomonas Lysosome-Related Organelles

S. Schmollinger, S. Chen, D. Strenkert, C. Hui, M. Ralle, S. S. Merchant

The paper uses XFM to quantify trace metal storage in Chlamydomonas acidocalcisomes under metal over-accumulation, showing Fe and Cu sequestration and distinct organelle populations.

bioRxiv · Feb 12, 2021Plant Biology

Hemozoin produced by mammals confers heme tolerance

R. Pek, X. Yuan, N. Rietzschel, J. Zhang, L. Jackson, E. Nishibori, A. Ribeiro, W. Simmons, J. Jagadeesh, H. Sugimoto, M. Z. Alam, L. Garrett, M. Haldar, M. Ralle, J. Phillips, , I. Hamza

The paper shows that mice lacking HRG1 accumulate excess heme in macrophages, which they tolerate by forming hemozoin, a process previously only observed in blood-feeding parasites.

bioRxiv · May 16, 2019Pathology