Author

M. S. da Silva

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

H. A. Armelin· 2 shared M. H. Dias· 2 shared A. C. Campi-Azevedo· 1 shared A. Chang· 1 shared A. Dekker· 1 shared A. Friskes· 1 shared A. Mulero-Sanchez· 1 shared A. Teixeira-Carvalho· 1 shared

Activity by Year

2017-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCancer Biology

H. A. Armelin

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Paradoxical activation of oncogenic signaling as a cancer treatment strategy

M. H. Dias, A. Friskes, S. Wang, J. M. H. Fernandes Neto, F. v. Gemert, S. Mourragui, H. J. Kuiken, S. Mainardi, D. Alvarez-Villanueva, C. Lieftink, B. Morris, A. Dekker, E. v. Dijk, C. Papagianni, M. S. da Silva, , R. Bernards

This study shows that inhibiting PP2A and WEE1 together can kill colon cancer cells by causing DNA replication collapse and cell death, and that resistance to this treatment suppresses tumor growth.

bioRxiv · Feb 6, 2023Cancer Biology

Fibroblast Growth Factor 2 lethally sensitizes cancer cells to stress-targeted therapeutic inhibitors

M. H. Dias, C. S. Fonseca, J. D. Zeidler, L. L. Albuquerque, M. S. da Silva, E. Cararo-Lopes, M. S. Reis, V. Noel, I. A. Prior, H. A. Armelin

This study shows that FGF2 sensitizes cancer cells to stress-targeted therapies by disrupting homeostasis and inducing synthetic lethal interactions with proteasome or DNA damage inhibitors.

Molecular Oncology · Dec 3, 2018Peer-reviewedCancer Biology

Analysis of the immunological biomarker profile during acute Zika virus infection reveals the overexpression of CXCL10, a chemokine already linked to neuronal damage

F. G. Naveca, G. Pontes, A. Chang, G. A. V. da Silva, V. A. do Nascimento, D. C. d. S. Monteiro, M. S. da Silva, L. F. Abdalla, J. H. A. Santos, T. A. P. de Almeida, M. d. C. C. Mejia, T. G. R. de Mesquita, H. V. d. S. Encarnacao, M. de Souza Gomes, L. R. Amaral, , R. Ramasawmy

This study identifies CXCL10 as a key biomarker of acute Zika virus infection through analysis of cytokine and chemokine levels in infected patients.

bioRxiv · Sep 8, 2017Immunology