Author

M. T. Lewno

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

H. Su· 2 shared J. Li· 2 shared J. Liu· 2 shared P. Wu· 2 shared X. Wang· 2 shared B. Pan· 1 shared C. Wang· 1 shared J. Fang· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersCell Biology

H. Su

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2 papers

Recent·Most cited

The COP9 Signalosome Suppresses Cardiomyocyte Necroptosis

P. Xiao, C. Wang, M. T. Lewno, P. Wu, J. Li, H. Su, J. O. Sternburg, J. Liu, X. Wang

This study shows that Cops8/CSN deficiency in cardiomyocytes leads to necroptosis via the RIPK1-RIPK3 pathway, with Nrf2 and redox stress playing key roles.

Circulation: Heart Failure · Aug 1, 2020Peer-reviewedCell Biology

The Calcineurin-TFEB-p62 Pathway Mediates the Activation of Cardiac Macroautophagy by Proteasomal Malfunction

B. Pan, N. Parajuli, Z. Tian, J. Li, P. Wu, M. T. Lewno, L. Bedford, R. J. Mayer, J. Fang, J. Liu, T. Cui, H. Su, X. Wang

This study identifies the Mocln1-calcineurin-TFEB-p62 pathway as mediating cardiac macroautophagy activation due to proteasomal malfunction.

Circulation Research · Jul 31, 2020Peer-reviewedCell Biology