Author

M. T. Lindenmeyer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

T. B. Huber· 2 shared A. Borchers· 1 shared A. Kaffke· 1 shared A. M. Mandel· 1 shared A. Peters· 1 shared A. Sivayoganathan· 1 shared B. Becher· 1 shared B. Schermer· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersImmunology

T. B. Huber

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2 papers

Recent·Most cited

ImmunologybioRxivJun 13, 2022

GM-CSF drives immune-mediated glomerular disease by licensing monocyte-derived cells to produce MMP12

H.-J. Paust, N. Song, D. DeFeo…·U. P. Panzer

The paper identifies GM-CSF-producing T cells that drive glomerulonephritis by licensing monocyte-derived cells to produce MMP12, leading to renal tissue damage.

DOI 10.1126/scitranslmed.add6137

humanmouse

𝕏 PostBluesky

Cell BiologybioRxivPreprintSep 13, 2020

Loss of genome maintenance accelerates podocyte damage

F. Braun, A. M. Mandel, L. Blomberg…·C. E. Kurschat

This study shows that loss of DNA repair gene Ercc1 in podocytes leads to DNA damage, proteinuria, and kidney disease, linked to mTORC1 activation and aging.

DOI 10.1101/2020.09.13.295303

mousehuman

𝕏 PostBluesky