Author

Marianna Lekakis

Division of Hematology, Department of Medicine, University of Miami Miller School of Medicine, Miami, Fl, USA;

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:mouse· 1

Frequent Co-Authors

Abdessamad Alaoui YoussfiiD· 2 shared Caroline Alice CoughliniD· 2 shared Daniel Bilbao CortesiD· 2 shared Dhanvantri ChahariD· 2 shared Francesco MauraiD· 2 shared Jonathan Harry SchatziD· 2 shared Alicia Bilbao-Martinez· 1 shared Anna Kingham· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

Abdessamad Alaoui YoussfiORCID

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2 papers

Recent·Most cited

BCL10 Gain-of-Function Impairs Germinal Center Participation by Naive B Cells and Cooperates with BCL6 to Drive Extrafollicular Diffuse Large B-Cell Lymphoma

Dhanvantri Chahar, Abdessamad Alaoui Youssfi, Caroline Alice Coughlin, Hsuan-Ting Huang, Vivian L.S. Kuan, Venu Venkatarame Gowda Saralamma, Marianna Lekakis, Dylan Huynh, Francesco Maura, Catalina Amador, Daniel Bilbao Cortes, Jayanta Chaudhuri, Jonathan H Schatz

This study shows BCL10 gain-of-function mutations prevent naive B cells from entering germinal centers and cooperate with BCL6 to cause extrafollicular diffuse large B-cell lymphoma.

bioRxiv · Nov 5, 2026 · PreprintCancer Biology

The cyclin-G associated kinase (GAK) is a novel mitotic kinase and therapeutic target in diffuse large B-cell lymphoma

Olivia Barbara Farag, Bassel Awada, Abdessamad Youssfi Alaoui, Paola Manara, Anna Kingham, Preet Kumar, Lingxiao Li, Brian B. Silver, Toral Shastri, Santiago Vilar, Austin D. Newsam, Marianna Lekakis, Diego E. Hernandez Parets, Pierre A. Younes, Dhanvantri Chahar, , Jonathan Harry Schatz

This paper identifies GAK as a novel mitotic kinase and therapeutic target in DLBCL, showing that GAK inhibition causes mitotic defects and that existing drugs can be repurposed for its targeting.

Leukemia · Nov 2, 2026Peer-reviewedCancer Biology