Author

Meike Soerensen

Max Planck Institute for Infection Biology

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Alexander KarlasiD· 1 shared Alexandra HrdinaiD· 1 shared Alla ZamyatinaiD· 1 shared André P. Mäurer· 1 shared Bastian PasiekaiD· 1 shared Carey D. NadelliD· 1 shared Cătălina Suzana StînguiD· 1 shared Cindy Rechner· 1 shared

Activity by Year

2017-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperMolecular Biology

Alexander KarlasORCID

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2 papers

Recent·Most cited

Colonization, translocation, and evolution of opportunistic pathogens during healthcare-associated infections

Martin Fenk, Alexandra Hrdina, James B. Winans, Meike Soerensen, Laura Ostertag, Elina Coquery, Fatoumata Ba Sow, Sirak Petros, Cătălina Suzana Stîngu, Norman Lippmann, Carey D. Nadell, Igor Iatsenko, Bastian Pasieka, Felix M. Key

The paper tracks the colonization and evolution of opportunistic pathogens in critically ill patients, identifying a mutation linked to healthcare-associated infections.

bioRxiv · Oct 23, 2025 · PreprintMicrobiology

ALPK1 And TIFA Dependent Innate Immune Response Triggered By The Helicobacter Pylori Type IV Secretion System

Stephanie Zimmermann, Lennart Pfannkuch, Munir A. Al‐Zeer, Sina Bartfeld, Manuel Koch, Jianping Liu, Cindy Rechner, Meike Soerensen, Olga V. Sokolova, Alla Zamyatina, Paul Kosma, André P. Mäurer, Frithjof Glowinski, Klaus‐Peter Pleißner, Monika Schmid, , Thomas F. Meyer

The paper identifies ALPK1 and TIFA as key components of an innate immune response triggered by H. pylori's T4SS, independent of CagA translocation.

Cell Reports · Sep 1, 2017Peer-reviewedMolecular Biology