Author

N. D. Christman

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:bacteria (other)· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Assay:metagenomics· 1 Organism:mouse· 1

Frequent Co-Authors

A. B. Dalia· 1 shared C. K. Ellison· 1 shared D. Erickson· 1 shared D. L. Erickson· 1 shared E. Wilson· 1 shared H. Eddington· 1 shared H. Q. Hughes· 1 shared J. Craft· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperMicrobiology

A. B. Dalia

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The PilT retraction ATPase promotes both extension and retraction of the MSHA type IVa pilus in Vibrio cholerae

H. Q. Hughes, N. D. Christman, T. N. Dalia, C. K. Ellison, A. B. Dalia

This study shows that PilT promotes both extension and retraction of MSHA type IVa pili in Vibrio cholerae, challenging the conventional understanding of T4aP dynamics.

bioRxiv · May 31, 2022 · PreprintMicrobiology

Increased microbial diversity and decreased prevalence of common pathogens in the gut microbiomes of wild turkeys compared to domestic turkeys

J. Craft, N. D. Christman, H. Eddington, J. M. Chaston, D. Erickson, E. Wilson

This study compares the gut microbiota of wild and domestic turkeys, finding higher microbial diversity and lower prevalence of common pathogens in wild turkeys.

bioRxiv · Jul 16, 2021Microbiology

A trimeric autotransporter enhances biofilm cohesiveness in Yersinia pseudotuberculosis but not in Yersinia pestis

J. T. Calder, N. D. Christman, J. M. Hawkins, D. L. Erickson

This study identifies a trimeric autotransporter, YadE, that enhances biofilm stability in Yersinia pseudotuberculosis but is inactivated in Yersinia pestis, likely due to incompatibility with Hms-ECM production.

bioRxiv · Mar 31, 2020Microbiology