Author

N. Skvir

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

A. A. Wilson· 1 shared C. Torres-Arancivia· 1 shared D. C. Liberti· 1 shared D. N. Kotton· 1 shared G. J. Murphy· 1 shared J. D. Rosarda· 1 shared J. K. Diedrich· 1 shared J. R. Yates· 1 shared

Activity by Year

2019-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

A. A. Wilson

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1 paper

Recent·Most cited

Amyloidogenic Proteins Drive Hepatic Proteostasis Remodeling in an Induced Pluripotent Stem Cell Model of Systemic Amyloid Disease

R. M. Giadone, D. C. Liberti, T. M. Matte, J. D. Rosarda, C. Torres-Arancivia, S. Ghosh, J. K. Diedrich, S. Pankow, N. Skvir, J.-C. Jean, J. R. Yates, A. A. Wilson, L. H. Connors, D. N. Kotton, R. L. Wiseman, G. J. Murphy

This study uses iPSC-derived hepatic cells to show that liver proteostasis factors and UPR signaling correlate with TTR amyloid production and can reduce amyloid secretion through ER stress-independent activation.

bioRxiv · Nov 10, 2019 · PreprintCell Biology