Author

Nadeen Taqatqa

Hebrew University of Jerusalem

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Molecular Biology· 2 Genomics· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

Eden EngaliD· 3 shared Gillian KayiD· 3 shared Maayan SaltoniD· 3 shared Mercedes Bentata· 3 shared Aveksha SharmaiD· 2 shared Mai BakeriD· 2 shared Ophir GeminderiD· 2 shared Sara Dahan· 2 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersMolecular Biology

Eden EngalORCID

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Author profile →

3 papers

Recent·Most cited

SMCHD1 loss triggers DUX4 expression by disrupting splicing in FSHD2

Eden Engal, Aveksha Sharma, Uria Aviel, Nadeen Taqatqa, Sarah Juster, Shiri Jaffe‐Herman, Mercedes Bentata, Ophir Geminder, Adi Gershon, Reyut Lewis, Gillian Kay, Merav Hecht, Silvina Epsztejn‐Litman, Marc Gotkine, Vincent Mouly, , Yotam Drier

This study shows SMCHD1 loss causes DUX4 expression in FSHD2 by disrupting splicing of DNMT3B, leading to hypomethylation and muscle abnormalities.

Science Advances · May 29, 2024Peer-reviewedGenomics

Isoforms of the TAL1 transcription factor have different roles in hematopoiesis and cell growth

Aveksha Sharma, Shani Mistriel-Zerbib, Rauf Ahmad Najar, Eden Engal, Mercedes Bentata, Nadeen Taqatqa, Sara Dahan, Klil Cohen, Shiri Jaffe‐Herman, Ophir Geminder, Mai Baker, Yuval Nevo, Inbar Plaschkes, Gillian Kay, Yotam Drier, , Maayan Salton

This study investigates the roles of TAL1 isoforms in hematopoiesis and cell growth, finding that TAL1-short promotes apoptosis and can act as a tumor suppressor.

PLoS Biology · Jun 28, 2023Peer-reviewedMolecular Biology

KDM3A regulates alternative splicing of cell-cycle genes following DNA damage

Mai Baker, Mayra Petasny, Nadeen Taqatqa, Mercedes Bentata, Gillian Kay, Eden Engal, Yuval Nevo, Ahmad Siam, Sara Dahan, Maayan Salton

This study shows KDM3A regulates alternative splicing of cell-cycle genes after DNA damage through phosphorylation and interaction with splicing factors.

RNA · Jul 28, 2021Peer-reviewedMolecular Biology