Author

Nataša Stanić

University of Southern Denmark

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Molecular Biology· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Approach:experimental assay· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Jan‐Wilhelm KornfeldiD· 3 shared Hande TopeliD· 2 shared Philip M. M. RuppertiD· 2 shared Aleksandra TrifunovićiD· 1 shared Ali AltıntaşiD· 1 shared Ana Rita TavaneziD· 1 shared Andrea Livia Silva RochaiD· 1 shared Anita ÖstiD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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3 shared papersMolecular Biology

Jan‐Wilhelm KornfeldORCID

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3 papers

Recent·Most cited

Molecular BiologyBMC Research NotesPeer-reviewedMay 6, 2026

EctoShed: A novel Gene Delivery Platform for Functional Analysis of Adipocyte-Shed Proteoforms

Ana Rita Tavanez, Nadia Meincke Egedal, Nataša Stanić…·Jan‐Wilhelm Kornfeld

EctoShed is a gene delivery platform that enables expression of both membrane-bound and soluble proteoforms in adipocytes, revealing their functional roles in metabolic regulation.

DOI 10.1186/s13104-026-07680-2

mouseproteomics

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Molecular BiologyeLifePeer-reviewedMar 17, 2026

Dietary sulfur amino acid restriction elicits a cold-like transcriptional response in inguinal but not epididymal white adipose tissue of male mice

Philip M. M. Ruppert, Aylin Seren Güller, Marcus Skjæveland…·Jan‐Wilhelm Kornfeld

The paper examines how dietary sulfur amino acid restriction and cold exposure affect energy expenditure and transcriptomic responses in different adipose tissues of male mice.

DOI 10.7554/elife.108825

mousebulk RNA-seq

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PhysiologyNature CommunicationsPeer-reviewedFeb 24, 2026

Male obesity causes adipose mitochondrial dysfunction in F1 progeny via a let-7-DICER axis.

Chien Hsiang Huang, Joo-Hyun Park, Ali Altıntaş…·Jan‐Wilhelm Kornfeld

This study shows that male obesity affects adipose mitochondrial function in offspring via the let-7-DICER axis, with reversible effects observed in both mice and human semen.

5 citationstop 1%

mousehuman

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