Author

Natalie C. Lamar

Pacific Northwest National Laboratory

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioinformatics· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Adam S. Cockrell· 1 shared Aleksey Y. OgurtsoviD· 1 shared Alexandra SchäferiD· 1 shared Amy María SimsiD· 1 shared Anthony S. BarenteiD· 1 shared Carrie D. Niccora· 1 shared Dustin L. Crockett· 1 shared Eric D. MerkleyiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperMicrobiology

Adam S. Cockrell

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

MARLOWE: Taxonomic Characterization of Unknown Samples for Forensics Using De Novo Peptide Identification

Sarah C. Jenson, Fanny Chu, Gelio Alves, Aleksey Y. Ogurtsov, Anthony S. Barente, Dustin L. Crockett, Natalie C. Lamar, Eric D. Merkley, Yi‐Kuo Yu, Kristin H. Jarman

MARLOWE is a computational tool for taxonomic characterization of unknown forensic samples using de novo peptide identification, achieving high specificity in species-level identification.

Scientific Reports · Aug 20, 2026Peer-reviewedBioinformatics

Dysregulation of lung epithelial cell homeostasis and immunity contributes to Middle East Respiratory Syndrome coronavirus disease severity

Amy María Sims, Alexandra Schäfer, Kenichi Okuda, Sarah R. Leist, Jacob F. Kocher, Adam S. Cockrell, Padraig E. Hawkins, Minako Furusho, Kara L. Jensen, Jennifer E. Kyle, Kristin E. Burnum-Johnson, Kelly G. Stratton, Natalie C. Lamar, Carrie D. Niccora, Karl K. Weitz, , Timothy Patrick Sheahan

This study uses multi-omics analysis to show that dysregulated lung epithelial cell homeostasis and immune responses drive severe MERS-CoV disease in mice.

mSphere · Jan 30, 2025Peer-reviewedMicrobiology