Author

O. Kanisicak

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Approach:genetic perturbation / CRISPR· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Gozdiff· 1 shared A. R. Guarnieri· 1 shared C. Paul· 1 shared G.-C. Fan· 1 shared J. B. Benoit· 1 shared J. Liang· 1 shared L. C. Green· 1 shared M. L. Nieman· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPhysiology

A. Gozdiff

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Author profile →

2 papers

Recent·Most cited

Myocardial IGF2R is a critical mediator of inflammation and fibrosis after ischemia-reperfusion injury

Z. Wu, W. Huang, X. He, S. Dutta, C. Paul, G.-C. Fan, O. Kanisicak, M. Xu, J. Liang, Y. Wang

This study investigates the role of IGF2R in myocardial ischemia-reperfusion injury, finding it promotes inflammation and fibrosis and is a potential therapeutic target.

bioRxiv · Apr 21, 2023 · PreprintCell Biology

Adipocyte deletion of the RNA binding protein HuR induces cardiac hypertrophy and fibrosis

A. R. Guarnieri, S. R. Anthony, A. Gozdiff, L. C. Green, S. Slone, M. L. Nieman, P. Alam, J. B. Benoit, O. Kanisicak, M. Tranter

This study shows that deleting HuR in adipose tissue causes cardiac hypertrophy and fibrosis, likely through inflammation and vesicle transport changes in subcutaneous white adipose tissue.

bioRxiv · Jan 19, 2021Physiology