Author

Oscar Maiques

Queen Mary University of London

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Abigail Lee· 1 shared Adrian BarrenoiD· 1 shared Andrea Garcia-Perez· 1 shared Andrejs BrauniD· 1 shared Andrew Clear· 1 shared Angel Delgado-Lopez· 1 shared Anna MaciàiD· 1 shared Christoph Cremer· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

Abigail Lee

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Lamin B1 Safeguards the B Cell Genome and Shapes Lymphoma Outcome

Filip Filipsky, Katarina B. Chapman, Johannes Bloehdorn, Oscar Maiques, Abigail Lee, Andrew Clear, Jun Wang, John G. Gribben, Michael Hausmann, Christoph Cremer, Andrejs Braun, Marta Crespi Sallan, Tetyana Klymenko

This study shows that Lamin B1 protects B cell genomes by preventing DNA damage and regulating chromatin accessibility, with its loss linked to poor lymphoma outcomes.

HemaSphere · Jun 1, 2026Peer-reviewedCancer Biology

Early overactivation of non-muscle myosin II during adaptation to combined BRAF and MEK inhibitors in dedifferentiating cutaneous melanomas

Andrea Garcia-Perez, Lucía Sanchez-Garcia, Marta Duran-Renieblas, Federica Sella, Silvia Novo-Acedo, Angel Delgado-Lopez, Adrian Barreno, Judith Gracia, Christopher Rios, Erica J. Young, Laszlo Radnai, Clara Diaz-Utrilla, Julia M. Martinez-Gomez, Rosa M. Marti, Marta C. Sallan, , José Luis Orgaz

This study shows that non-muscle myosin II overactivation occurs in dedifferentiating melanomas adapting to BRAF and MEK inhibitors, and targeting NMII with BMi reduces drug-tolerant persister cell survival.

bioRxiv · Dec 3, 2025 · PreprintCancer Biology