Author

Filip Filipsky

University of BaselORCID 0000-0002-4357-4464Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

Heinz Philipp LaubliiD· 2 shared Abigail Lee· 1 shared Amanda Carlos Paulino· 1 shared Andreas Wilfer· 1 shared Andreas ZinggiD· 1 shared Andrejs BrauniD· 1 shared Andrew Clear· 1 shared Anne EugsteriD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersCancer Biology

Heinz Philipp LaubliORCID

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3 papers

Recent·Most cited

Targeting poorly differentiated thyroid cancer with bispecific antibodies and CAR T cells from the human anti-TSHR autoantibody repertoire

Claudia Fischer, Anne Eugster, Brenda Besemer, Mélanie Buchi, Marcus Bauer, Christoph Schultheiß, Elisa D’Arcangelo, Filip Filipsky, Claudia Wickenhauser, Andreas Wilfer, Kerstin Lorenz, Jens Jakscha, Laurent Müller, Roswitha Köberle-Wührer, Benjamin Kasenda, , Mascha Binder

This paper explores using anti-TSHR autoantibodies to create bispecific antibodies and CAR T cells for treating poorly differentiated thyroid cancer, showing potent in vitro and in vivo activity.

bioRxiv · Oct 20, 2026 · PreprintCancer Biology

Lamin B1 Safeguards the B Cell Genome and Shapes Lymphoma Outcome

Filip Filipsky, Katarina B. Chapman, Johannes Bloehdorn, Oscar Maiques, Abigail Lee, Andrew Clear, Jun Wang, John G. Gribben, Michael Hausmann, Christoph Cremer, Andrejs Braun, Marta Crespi Sallan, Tetyana Klymenko

This study shows that Lamin B1 protects B cell genomes by preventing DNA damage and regulating chromatin accessibility, with its loss linked to poor lymphoma outcomes.

HemaSphere · Jun 1, 2026Peer-reviewedCancer Biology

Local glycan engineering induces systemic antitumor immune reactions via antigen cross-presentation

Natália Rodrigues Mantuano, Michael T. Sandholzer, Emiel Rossing, Johan F. A. Pijnenborg, Andreas Zingg, Filip Filipsky, Ronja Wieboldt, Amanda Carlos Paulino, Isabelle V. M. Siqueira, Thomas Jan Boltje, Heinz Philipp Laubli

This study shows that local glycan engineering using AAV-delivered sialidases reduces tumor growth and enhances systemic antitumor immunity through improved cross-presentation and dendritic cell activation.

bioRxiv · May 7, 2026 · PreprintImmunology