Author

P.-M. Coly

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Assay:multiome· 1

Frequent Co-Authors

S. Chatterjee· 2 shared C. Boulanger· 1 shared C. Devue· 1 shared C. El Jekmek· 1 shared C. M. Boulanger· 1 shared D. Loew· 1 shared F. Dingli· 1 shared F. Mezine· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

S. Chatterjee

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Author profile →

2 papers

Recent·Most cited

Atheroprone shear stress stimulates noxious endothelial extracellular vesicle uptake by MCAM and PECAM-1 cell adhesion molecules

P.-M. Coly, S. Chatterjee, F. Mezine, C. El Jekmek, C. Devue, T. Nipoti, M. Lara Corona, F. Dingli, D. Loew, G. van Niel, C. M. Boulanger

This study investigates how atheroprone and atheroprotective shear stress affects endothelial extracellular vesicle (EV) release and uptake, finding that LSS increases EV uptake through adhesion molecules like MCAM and PECAM-1.

bioRxiv · Dec 31, 2022 · PreprintCell Biology

Restoring vascular endothelial autophagic flux reduces atherosclerotic lesions

S. Chatterjee, M. Kheloufi, S. M. I. Mazlan, X. Loyer, T. A. McKinsey, C. Boulanger, P.-M. Coly

This study shows that inhibiting HDAC6 restores autophagic flux in endothelial cells under low shear stress, reducing atherosclerotic lesion formation.

bioRxiv · May 6, 2021 · PreprintPathology