Interleukin-6 is critical in the development of gcn2-mutation associated pulmonary vascular disease in mice
Max Schwiening, Qingyue Gao, Mark R. Southwood, Alexi Crosby, Stephen Moore, José Antonio Valer, Niki Veale, Benjamin J. Dunmore, Paul D. Upton, Alfred A Roger Thompson, Nicholas W. Morrell, Stefan John Marciniak, Elaine Soon
This study shows that interleukin-6 is critical in gcn2-mutation associated pulmonary vascular disease in mice, as its deletion rescues pulmonary hypertension phenotypes.
bioRxiv · Apr 14, 2025 · PreprintPathology