Author

R. C. Glover

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Organism:bacteria (other)· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Approach:genetic perturbation / CRISPR· 1 Assay:metagenomics· 1

Frequent Co-Authors

M. K. Thomason· 2 shared M. L. Reniere· 2 shared A. J. Olive· 1 shared A. J. Pollock· 1 shared A. P. McFarland· 1 shared C. R. Halsey· 1 shared H. Tabakh· 1 shared J. J. Woodward· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersMicrobiology

M. K. Thomason

Mutual co-authors in cluster

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3 papers

Recent·Most cited

A genome-wide screen in macrophages identifies PTEN as required for myeloid restriction of Listeria monocytogenes infection

R. C. Glover, N. H. Schwardt, S.-K. E. Leano, M. E. Sanchez, M. K. Thomason, A. J. Olive, M. L. Reniere

This study identifies PTEN as a host factor required for macrophage phagocytosis of Listeria monocytogenes, revealing its role in bacterial adherence and host protection during infection.

bioRxiv · Dec 12, 2022 · PreprintMicrobiology

The global redox-responsive transcriptional regulator Rex represses fermentative metabolism and is required for Listeria monocytogenes pathogenesis

C. R. Halsey, M. K. Thomason, R. C. Glover, M. L. Reniere

This study shows that Rex, a redox-responsive regulator in L. monocytogenes, represses fermentative metabolism and is required for bacterial survival in the gallbladder during listeriosis.

bioRxiv · Feb 16, 2021Microbiology

4-hydroxy-2-nonenal antimicrobial toxicity is neutralized by an intracellular pathogen.

H. Tabakh, A. P. McFarland, A. J. Pollock, R. C. Glover, J. J. Woodward

The paper shows that 4-HNE, a product of ROS during infection, is counteracted by L. monocytogenes through rha1/2 genes, enhancing bacterial survival.

bioRxiv · May 25, 2020Microbiology