Cancer BiologybioRxivPreprint
ATE1 activates ER-stress and UPR pathways in glioblastoma
J. Macedo-da-Silva, S. M. Oba-Shinjo, L. Rosa-Fernandes…·G. Palmisano
This study investigates the role of ATE1-mediated N-terminal arginylation in glioblastoma, finding it activates ER-stress and UPR pathways, promotes autophagy, and supports tumor growth.
DOI 10.1101/2023.09.25.559258