Author

R. Jühlen

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1 Organism:mouse· 1

Frequent Co-Authors

D. Landgraf· 2 shared A. Hübner· 1 shared A. Huebner· 1 shared G. Eisenhofer· 1 shared K. Koehler· 1 shared K. Köhler· 1 shared M. Peitzsch· 1 shared S. Gärtner· 1 shared

Activity by Year

2017-2018 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 18 mutual co-author links

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2 shared papersCell Biology

D. Landgraf

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2 papers

Recent·Most cited

Cell BiologybioRxivJul 31, 2018

Triple A patient cells suffering from mitotic defects fail to localize PGRMC1 to mitotic kinetochore fibers

R. Jühlen, D. Landgraf, A. Hübner, K. Köhler

The paper shows that ALADIN depletion causes mislocalization of Aurora A and PGRMC1, and overexpression of PGRMC2 in triple A syndrome fibroblasts, leading to mitotic errors.

DOI 10.1186/s13008-018-0041-5

humanproteomics

𝕏 PostBluesky

Cell BiologybioRxivJul 17, 2017

Compensation For Chronic Oxidative Stress In ALADIN Null Mice

R. Jühlen, M. Peitzsch, S. Gärtner…·K. Koehler

This study shows ALADIN null mice do not develop a triple A-like phenotype under chronic oxidative stress, indicating a compensatory mechanism in glutathione metabolism.

DOI 10.1242/bio.030742

mousemetabolomics

𝕏 PostBluesky