R. M. H. Merks

The paper models cell-ECM mechanical interactions using hybrid cellular Potts and molecular dynamics methods, showing force propagation and network stiffening consistent with experiments.

The paper uses a hybrid cellular Potts model to study how stress fiber orientation affects traction forces in cells on micropatterned substrates, reproducing experimental data on fibroblasts, endothelial, and epithelial cells.

This paper develops a computational model to simulate how extracellular matrix composition affects lymphocyte motility patterns through cell-matrix adhesion dynamics.

This paper shows that genome architecture evolves to enable mutation-driven division of labor in bacteria by separating growth and antibiotic genes with fragile sites.

The paper proposes a mechanism for epithelial branching morphogenesis without proliferation or mesenchymal interaction, showing autocrine TGFβ1 inhibits protrusions and drives branching through curvature-dependent inhibition.

This study shows that multicellular aggregates evolve due to more efficient chemotaxis, but unicellular states emerge in rapidly changing environments through dispersal, creating evolutionary bi-stability.

The paper develops a computational model of cambium activity to simulate radial plant growth by integrating regulatory factors and physical constraints.