Author

R. Zeillinger

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:imaging· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

A. E. Dick· 1 shared A. Vanderstichele· 1 shared C. C. Valentine· 1 shared D. Nachmanson· 1 shared D. Slade· 1 shared D. W. Gerlich· 1 shared E. Kukolj· 1 shared E. Loter· 1 shared

Activity by Year

2017-2018 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

A. E. Dick

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Author profile →

2 papers

Recent·Most cited

Ultra-sensitive sequencing for cancer detection reveals progressive clonal selection in normal tissue over a century of human lifespan

J. J. Salk, K. Loubet-Senear, E. Maritschnegg, C. C. Valentine, L. N. Williams, R. Horvat, A. Vanderstichele, D. Nachmanson, K. T. Baker, M. J. Emond, E. Loter, T. Soussi, L. A. Loeb, R. Zeillinger, P. Speiser, R. A. Risques

This study uses ultra-sensitive sequencing to detect TP53 mutations in normal tissues across age, showing that age-related mutations share clonal selection traits with tumor mutations.

bioRxiv · Nov 4, 2018Cancer Biology

PARP inhibition causes premature loss of cohesion in cancer cells

E. Kukolj, T. Kaufmann, A. E. Dick, R. Zeillinger, D. W. Gerlich, D. Slade

This study shows PARP inhibition causes premature cohesion loss in cancer cells during mitosis, leading to sister chromatid scattering, and identifies PARP1/2 entrapment at replication forks as a mechanism.

bioRxiv · Jun 22, 2017Cell Biology