Author

Rebecca Lancaster

Queen's University Belfast

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Assay:proteomics· 3 Organism:bacteria (other)· 2 Research Focus:cell signaling & trafficking· 2 Approach:biophysical modeling· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1

Frequent Co-Authors

José Antonio BengoecheaiD· 3 shared Joana Sá‐PessoaiD· 2 shared Adrien Kissenpfenning· 1 shared Amy DumiganiD· 1 shared Charlotte BuckleyiD· 1 shared Ciara Ross· 1 shared Grant Mills· 1 shared Jan K. MarzinekiD· 1 shared

Activity by Year

2025-2025 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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3 shared papersMicrobiology

José Antonio BengoecheaORCID

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3 papers

Recent·Most cited

Klebsiella pneumoniae remodels its Kdo2-lipid A in a TLR4-dependent manner to adapt to the macrophage intracellular environment

Toby Leigh Bartholomew, Joana Sá‐Pessoa, Rebecca Lancaster, Nan Zhang, Karolina Wojtania, Steven J. Hancock, Grant Mills, Adrien Kissenpfenning, José Antonio Bengoechea

This study shows Klebsiella pneumoniae modifies its lipid A in response to macrophage environment, enhancing survival and immune evasion through TLR4-dependent mechanisms.

bioRxiv · Jun 20, 2025 · PreprintMicrobiology

Interrogating the genus Yersinia to define the rules of lipopolysaccharide lipid A structure associated with pathogenicity.

Maria Helena Fernandez-Carrasco, Käthe M. Dahlström, Ricardo Calderón-González, Amy Dumigan, Jan K. Marzinek, Rebecca Lancaster, Peter John Bond, Tiina Annamaria Salminen, José Antonio Bengoechea

The paper investigates how lipid A modifications in Yersinia species influence pathogenicity by altering TLR4 recognition and inflammation.

bioRxiv · Mar 3, 2025 · PreprintMicrobiology

Klebsiella pneumoniae disrupts vasodilation by targeting eNOS post translational modifications via the type VI secretion system and the capsule polysaccharide

Safi Rehman, Joana Sá‐Pessoa, Charlotte Buckley, Rebecca Lancaster, Ciara Ross, Xun Zhang, John G. McCarron, Tim M. Curtis, José Antonio Bengoechea

This study shows Klebsiella pneumoniae inhibits vasodilation by targeting eNOS post-translational modifications through its T6SS and capsule polysaccharide.

bioRxiv · Feb 5, 2025 · PreprintMicrobiology