Author

Richard Davenport

Washington University in St. Louis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genomics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Approach:machine learning (classical)· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Bruno A. BeníteziD· 2 shared Carlos CruchagaiD· 2 shared Fengxian WangiD· 2 shared John BuddeiD· 2 shared Jorge L. Del‐AguilaiD· 2 shared Joseph D. DoughertyiD· 2 shared Joseph L. BradleyiD· 2 shared Kathie A. MihindukulasuriyaiD· 2 shared

Activity by Year

2019-2019 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersGenomics

Bruno A. BenítezORCID

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2 papers

Recent·Most cited

A single- nuclei RNA sequencing study of Mendelian and sporadic AD in the human brain

Jorge L. Del‐Aguila, Zeran Li, Umber Dube, Kathie A. Mihindukulasuriya, John Budde, María Victoria Fernández, Laura Ibáñez, Joseph L. Bradley, Fengxian Wang, Kristy Bergmann, Richard Davenport, John C. Morris, David M. Holtzman, Richard J. Perrin, Bruno A. Benítez, , Oscar Harari

This study uses single-nuclei RNA-seq to compare cell proportions and gene expression in Mendelian and sporadic AD brains, identifying reduced excitatory neurons in a Mendelian carrier and proposing best practices for analysis.

Alzheimer s Research & Therapy · Aug 9, 2019Peer-reviewedGenomics

The TMEM106B rs1990621 protective variant is also associated with increased neuronal proportion

Zeran Li, Fabiana Geraldo Farias, Umber Dube, Jorge L. Del‐Aguila, Kathie A. Mihindukulasuriya, María Victoria Fernández, Laura Ibáñez, John Budde, Fengxian Wang, Allison M. Lake, Yuetiva K. Deming, James Perez, Chengran Yang, Joseph L. Bradley, Richard Davenport, , Carlos Cruchaga

The paper identifies a genetic variant in TMEM106B associated with increased neuronal proportion in neurodegenerative and cognitive normal cohorts, suggesting a role in neuronal protection.

bioRxiv · Mar 20, 2019 · PreprintNeuroscience