Author

Roman Shaposhnikov

University of Milano-Bicocca

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Alberto FerriiD· 1 shared Alice Campana· 1 shared Cristiana ValleiD· 1 shared Gabriella Gualtieri· 1 shared Illari SalvatoriiD· 1 shared Mara ZilocchiiD· 1 shared Roland NicsanuiD· 1 shared Silvia LombardiiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

Alberto FerriORCID

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1 paper

Recent·Most cited

Nuclear DNA damage is a primary driver of mitochondrial dysfunction in C9ORF72 ALS/FTD

Mara Zilocchi, Illari Salvatori, Silvia Lombardi, Roland Nicsanu, Alice Campana, Roman Shaposhnikov, Gabriella Gualtieri, Silvia Scaricamazza, Cristiana Valle, Alberto Ferri, Silvia M.L. Barabino

This study shows that nuclear DNA damage directly causes mitochondrial dysfunction in C9ORF72 ALS/FTD by triggering a DNA damage response that depletes NAD+ and impairs mitochondrial bioenergetics.

bioRxiv · May 21, 2026 · PreprintCell Biology