Author

Illari Salvatori

Fondazione Santa LuciaORCID 0000-0002-8397-8500Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Alberto FerriiD· 2 shared Cristiana ValleiD· 2 shared Silvia ScaricamazzaiD· 2 shared Alessandro MatteocciiD· 1 shared Alice Campana· 1 shared Daniela Francesca AngeliniiD· 1 shared Daniele Lettieri‐BarbatoiD· 1 shared Flaminia RiggioiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCell Biology

Alberto FerriORCID

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintMay 21, 2026

Nuclear DNA damage is a primary driver of mitochondrial dysfunction in C9ORF72 ALS/FTD

Mara Zilocchi, Illari Salvatori, Silvia Lombardi…·Silvia M.L. Barabino

This study shows that nuclear DNA damage directly causes mitochondrial dysfunction in C9ORF72 ALS/FTD by triggering a DNA damage response that depletes NAD+ and impairs mitochondrial bioenergetics.

DOI 10.64898/2026.05.20.726184

humanDNA methylation

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NeuroscienceHeliyonPeer-reviewedJan 22, 2025

Brown Adipose Tissue undergoes pathological perturbations and shapes C2C12 myoblast homeostasis in the SOD1-G93A mouse model of Amyotrophic Lateral Sclerosis.

Marco Rosina, Silvia Scaricamazza, Flaminia Riggio…·Alberto Ferri

The paper investigates changes in brown adipose tissue and its extracellular vesicles in an ALS mouse model, showing their impact on myoblast homeostasis and muscle atrophy.

3 citations

mousebulk RNA-seq

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