Author

S. C. Piatt

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Organism:E. coli· 1 Assay:imaging· 1

Frequent Co-Authors

A. T. Moreno· 1 shared E. S. Thrall· 1 shared F. W. Lopezcolorado· 1 shared J. J. Loparo· 1 shared J. Loparo· 1 shared J. M. Stark· 1 shared M. Cisneros-Aguirre· 1 shared S. Chang· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 18 mutual co-author links

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1 shared paperBiochemistry

A. T. Moreno

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2 papers

Recent·Most cited

Replication stalling activates SSB for recruitment of DNA damage tolerance factors

E. S. Thrall, S. C. Piatt, S. Chang, J. Loparo

This study shows that replication stress causes SSB to recruit TLS polymerase Pol IV and other SIPs to stalled forks, suggesting a general mechanism for repair factor enrichment.

bioRxiv · May 24, 2022 · PreprintBiochemistry

XLF acts as a flexible connector during non-homologous end joining

S. M. Carney, A. T. Moreno, S. C. Piatt, M. Cisneros-Aguirre, F. W. Lopezcolorado, J. M. Stark, J. J. Loparo

This study shows that the C-terminal tail of XLF is essential for DNA end alignment during NHEJ, with a minimal length required for efficient end joining.

bioRxiv · Jul 29, 2020Biochemistry