Author

S. de Taeye

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:proteomics· 2

Frequent Co-Authors

J. den Dunnen· 2 shared M. van Gils· 2 shared W. Hoepel· 2 shared A. Neele· 1 shared A. P. Bye· 1 shared A. Vlaar· 1 shared C. van Drunen· 1 shared F. Linty· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersImmunology

J. den Dunnen

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2 papers

Recent·Most cited

Aberrant glycosylation of anti-SARS-CoV-2 IgG is a pro-thrombotic stimulus for platelets

A. P. Bye, W. Hoepel, J. L. Mitchell, S. Jegouic, S. Loureiro, T. Sage, S. de Taeye, M. van Gils, N. Kriek, N. Cooper, I. Jones, J. den Dunnen, J. M. Gibbins

The paper shows that aberrant glycosylation of anti-SARS-CoV-2 IgG enhances platelet activation and thrombosis via FcyRIIA, which can be inhibited by tyrosine kinase inhibitors and P2Y12 antagonists.

bioRxiv · Mar 26, 2021Immunology

Anti-SARS-CoV-2 IgG from severely ill COVID-19 patients promotes macrophage hyper-inflammatory responses

W. Hoepel, H.-J. Chen, S. Allahverdiyeva, X. Manz, J. Aman, P. Bonta, P. Brouwer, S. de Taeye, T. Caniels, K. van der Straten, K. Golebski, G. Griffith, R. Jonkers, M. Larsen, F. Linty, , J. den Dunnen

Anti-SARS-CoV-2 IgG from severely ill COVID-19 patients induces hyper-inflammatory responses in macrophages, leading to endothelial barrier breakdown and microvascular thrombosis.

bioRxiv · Jul 13, 2020Immunology