Author

S. Gilja

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Approach:experimental assay· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

H. Levine· 2 shared J. A. Somarelli· 2 shared K. E. Ware· 2 shared M. K. Jolly· 2 shared A. J. Armstrong· 1 shared A. J. Hish· 1 shared A. L. Lazarides· 1 shared D. G. Gerber· 1 shared

Activity by Year

2017-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

H. Levine

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2 papers

Recent·Most cited

E-cadherin represses anchorage-independent growth in sarcomas through both signaling and mechanical mechanisms

M. K. Jolly, K. E. Ware, S. Xu, S. Gilja, S. Shetler, Y. Yang, X. Wang, G. Austin, D. Runyambo, A. J. Hish, S. Bartholf DeWitt, J. T. George, R. T. Kreulen, M.-K. Boss, A. L. Lazarides, , J. A. Somarelli

This study identifies E-cadherin's role in suppressing sarcoma growth through both signaling (inhibiting p-CREB and TBX2) and mechanical (enhancing cell-cell adhesion) mechanisms.

bioRxiv · Jan 10, 2019Cancer Biology

EMT and MET: necessary or permissive for metastasis?

M. K. Jolly, K. E. Ware, S. Gilja, J. A. Somarelli, H. Levine

This paper examines the role of EMT and MET in metastasis, arguing that these processes are not binary and proposes alternative mechanisms beyond the traditional EMT-MET model.

bioRxiv · Mar 29, 2017Cancer Biology