Author

S. K. Fey

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:mouse· 2

Frequent Co-Authors

A. D. Campbell· 2 shared A. K. Najumudeen· 2 shared C. A. Ford· 2 shared C. Nixon· 2 shared J. P. Morton· 2 shared K. Gilroy· 2 shared O. J. Sansom· 2 shared W. Clark· 2 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

A. D. Campbell

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

KRAS allelic imbalance drives tumour initiation yet suppresses metastasis in colorectal cancer in vivo

A. K. Najumudeen, S. K. Fey, L. M. Millet, C. A. Ford, K. Gilroy, N. Gunduz, R. A. Ridgway, E. Anderson, D. Strathdee, W. Clark, C. Nixon, J. P. Morton, A. D. Campbell, O. J. Sansom

This study shows that wild-type KRAS suppresses tumour growth and metastasis in KRAS mutant colorectal cancer while enhancing resistance to MEK inhibition.

bioRxiv · Aug 29, 2023 · PreprintCancer Biology

Kras loss of heterozygosity promotes MAPK dependent pancreatic ductal adenocarcinoma and induces therapeutic sensitivity

S. K. Fey, A. K. Najumudeen, C. A. Ford, K. Gilroy, R. Upstill-Goddard, W. Clark, C. Nixon, D. K. Chang, S. T. Barry, J. P. Morton, A. D. Campbell, O. J. Sansom

This study shows that loss of wild-type Kras enhances oncogenic Kras signaling and promotes pancreatic ductal adenocarcinoma, while also increasing sensitivity to MEK1/2 inhibitors.

bioRxiv · Aug 29, 2023 · PreprintCancer Biology