Author

S. Kissler

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3 Cell Biology· 1

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Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Approach:genetic perturbation / CRISPR· 2 Research Focus:cell type annotation· 1 Assay:flow cytometry· 1 Assay:metabolomics· 1

Frequent Co-Authors

K. Bode· 2 shared A. Messent· 1 shared B. Kiaf· 1 shared B. Lelli· 1 shared B. Weigelin· 1 shared C. M. Schuster· 1 shared C. Schuster· 1 shared D. J. Lefeber· 1 shared

Activity by Year

2017-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 11 mutual co-author links

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2 shared papersImmunology

K. Bode

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Beta Cells Deficient for Renalase Counteract Autoimmunity by Shaping Natural Killer Cell Activity

K. Bode, S. Wei, I. Gruber, S. Kissler, P. Yi

This study shows that beta cells lacking Renalase reduce autoimmunity by altering Natural Killer cell activity and enhancing anti-inflammatory responses.

bioRxiv · Feb 29, 2024 · PreprintImmunology

Gata3 is detrimental to regulatory T cell function in autoimmune diabetes

B. Kiaf, K. Bode, C. M. Schuster, S. Kissler

The paper shows that Gata3 in Tregs worsens autoimmune diabetes by reducing their suppressive function, and deleting Gata3 in Tregs protects against diabetes.

bioRxiv · Mar 18, 2023 · PreprintImmunology

Glycocalyx-mediated Cell Adhesion and Migration

S. Schmidt, B. Weigelin, J. te Riet, V. te Boekhorst, M. te Lindert, M. Wijers-Rouw, B. Lelli, L. Rognoni, M. Krause-Vortmeyer, A. Messent, L. Bracci, K. E. Gottschalk, S. Kissler, M. Humphries, D. J. Lefeber, , P. Friedl

This study shows the glycocalyx enables cell adhesion and amoeboid migration through nanoscale interactions and mechano-coupling, independent of integrin function.

bioRxiv · Jun 12, 2020 · PreprintCell Biology

Peripherally-induced regulatory T cells contribute to the control of autoimmune diabetes

C. Schuster, F. Zhao, S. Kissler

This study shows that peripherally-induced regulatory T cells (pTregs) are crucial for preventing autoimmune diabetes in NOD mice by modulating disease risk.

bioRxiv · Oct 7, 2017Immunology