Author

S. Sumitomo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Assay:DNA methylation· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Assay:scRNA-seq· 2 Approach:statistical model· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

A. Suzuki· 2 shared K. Fujio· 2 shared K. Ishigaki· 2 shared K. Yamamoto· 2 shared M. Ota· 2 shared Y. Kochi· 2 shared H. Inui· 1 shared H. Tsuchiya· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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2 shared papersImmunology

A. Suzuki

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2 papers

Recent·Most cited

Immune cell multi-omics analysis reveals contribution of oxidative phosphorylation to B cell functions and organ damage of lupus

Y. Takeshima, Y. Iwasaki, M. Nakano, Y. Narushima, M. Ota, Y. Nagafuchi, S. Sumitomo, T. Okamura, K. B. Elkon, K. Ishigaki, A. Suzuki, Y. Kochi, K. Yamamoto, K. Fujio

The paper identifies oxidative phosphorylation (OXPHOS) as a key driver in SLE B cells, linking mitochondrial dysfunction to immune signaling and organ damage.

bioRxiv · Oct 8, 2021 · PreprintImmunology

Parsing multiomics landscape of activated synovial fibroblasts highlights drug targets linked to genetic risk of rheumatoid arthritis

H. Tsuchiya, M. Ota, S. Sumitomo, K. Ishigaki, A. Suzuki, T. Sakata, Y. Tsuchida, H. Inui, J. Hirose, Y. Kochi, Y. Kadono, K. Shirahige, S. Tanaka, K. Yamamoto, K. Fujio

This study analyzes multi-omics data from activated synovial fibroblasts to identify genetic risk factors and potential drug targets for rheumatoid arthritis.

bioRxiv · Jun 3, 2020Immunology